Crafting an Optimal Peaty Androgenic/Anabolic Hormone Stack

metabolicmilk

Hi Everyone,

I'm on a quest to create the most anabolic/androgenic peaty stack. My goal is to stay true to some of @haidut's recommendations, as well as ensure the stack is well-tuned to his product formulations.

Dehydroepiandrosterone (DHEA): Capping at Haidut's recommended maximum of 15mg daily.

Pregnenolone: Keeping it at a 1:1 ratio with DHEA, which equates to 15mg.

Dihydrotestosterone (DHT)/Androsterone (Andro): Also at a 1:1 ratio with DHEA, resulting in 15mg.

Progesterone: Following a minimum 3:1 ratio with DHEA, thus I'm looking at 45mg.

I would then like to add ( not on @haidut's recommendation ) :
Testosterone (T): 30mg.

2 points to make :

The Testosterone dose might be adjusted to non-suppressive levels. ( ie 1 - 10mg )
Alternatively, Pregnenolone could be increased to roughly 5-10 times the T dose, as suggested by Haidut to avert potential shutdowns.

Here's where I'm seeking some help:
Would increasing Pregnenolone to 5-10 times the T dose lead to excessive DHEA or provoke any adverse effects? While I'm less concerned about its conversion to Progesterone, considering Haidut's stance that Progesterone can be utilized even in an 8:1 ratio with DHEA.

I'm curious as to any feedback and advice anyone is willing to share - or any similar experiences with such a stack

Mulloch94

I would probably just dissolve like 800mgs of DHEA into a bottle of Progest-E. That will give you roughly 4:1 to 5:1 ratio of progesterone to DHEA (my figures are ballparks based on the presumption there's around 3grams of P4 in a given bottle of progest-e. I recommend if you do this to be a little more precise in your measurements).

Rubbing it on your gums will give a good effect. There would be no need for pregnenolone in this stack. Since progesterone and DHEA are both precursors of testosterone you probably won't need T either. Of course adding stronger androgens will elicit a greater effect, but it wouldn't exactly fit the criteria of "peaty."

babou

@metabolicmilk Hello, I know its a slight necro but I'm wondering if you have gone ahead and tried what you describe here. I myself am also running a very similar experiment using pansterone, pregnenolone, and some tyromix in conjunction with small doses of DHT (5-10 mg a day) to see what happens, and especially curious about whether this stack will increase or decrease endogenous androgen production after removing DHT and eventually cycling off other components as well (I don't really want a hormone permastack the way you would do for vitamins, mostly I want to shift my body into a certain state and then use more gentle/natural things to sustain it)

lobotomize-me

sr 9011 looks interesting/peaty check this thread out https://lowtoxinforum.com/threads/stenabolic.27227/
a list of its benefits mentioned

All the effects observed for SR9009 derive from REV-ERB (NR1D1, NR1D2) activation in the body.

REV-ERB affects the circadian behavior of mice, altering their activity and sleep patterns [R].

Besides its effect on the circadian rhythm (suppression of BMAL1 production), REV-ERB affects many other functions related to energy production [R, R].

REV-ERB is mainly found in liver, muscle, and fat tissue:

Liver: REV-ERB affects the rhythm of 90% of the about 900 genes under circadian control in mice liver. It turns off the genes that produce glucose without altering insulin sensitivity. It also turns on genes that generate new fat cells and reduces the inflammatory response (by affecting macrophage production) [R, R, R, R].

Muscles: REV-ERB promotes the burning of fat, increases the activity of mitochondria, and promotes the generation of new while decreasing the destruction of old mitochondria [R].

Fat cells: REV-ERB turns off the genes responsible for storing fat and decreases triglyceride production [R].
In animal and cell studies, SR9009:

Increased oxygen consumption [R]

Decreased generation of new fat cells in the liver [R]

Decreased production of cholesterol and bile acids in the liver [R]

Increased the number of mitochondria in muscle [R]

Increased glucose and fatty acid use in muscle [R]

Decreased fat storage [R]