Fructose - Resources

[izkrov]

Looking to pool resources on the effects of fructose, I've never felt any different from eating it but some people it upsets their tummies. Ray's mentioned that fructose is also mitochondrial uncoupler

If someone knows a source for that or the reasoning behind it, please poast.
Anyway, here's some sources from his newsletters:

Fructose promotes sodium retention (https://pubmed.ncbi.nlm.nih.gov/6344611/)
Diabetics have increased vascular leakiness (https://pubmed.ncbi.nlm.nih.gov/12564648)
Fructose lowers blood vessel leakiness (https://pubmed.ncbi.nlm.nih.gov/14553836/)
And the "Sugar Issues" newsletter from 2011 talks about how you can use fructose when glucose isn't available, as well as eating more calories but retaining the same weight.

Some Haidut sources. He has a study that shows fructose didn't deplete liver atp which is a main argument against fructose
https://twitter.com/haidut/status/1751765657648038132

[Fructose]

@izkrov said in Fructose - Resources:

Looking to pool resources on the effects of fructose, I've never felt any different from eating it but some people it upsets their tummies. Ray's mentioned that fructose is also mitochondrial uncoupler

If someone knows a source for that or the reasoning behind it, please poast.
Anyway, here's some sources from his newsletters:

Fructose promotes sodium retention (https://pubmed.ncbi.nlm.nih.gov/6344611/)
Diabetics have increased vascular leakiness (https://pubmed.ncbi.nlm.nih.gov/12564648)
Fructose lowers blood vessel leakiness (https://pubmed.ncbi.nlm.nih.gov/14553836/)
And the "Sugar Issues" newsletter from 2011 talks about how you can use fructose when glucose isn't available, as well as eating more calories but retaining the same weight.

Some Haidut sources. He has a study that shows fructose didn't deplete liver atp which is a main argument against fructose
https://twitter.com/haidut/status/1751765657648038132

Perhaps fructose, as an insulin mimetic, has a similar effect to insulin on the oxidation of fatty acids, which may contribute to better oxidative phosphorylation.