Ultra Low fat Diet Logs <20g (mostly saturated)
-
Recently I’ve been trying to go low fats. Under 30 grams ish.
I’ve been drinking OJ with nicinamide and salt, some milk, a low fat cheese (4g per 8g of protein), ham (yes I know nitrates are bad but the vitamin c helps with thag), mangos, watermelon, mangosteen, etc, lots of raw agave syrup- I wanted to use honey but agave had more quantity for cheap-, and very lean chicken breast and beef and bone broth.
I felt amazing initially bht my bowel minvents have started to slow down immediately.
Each time I drink milk or eat fat however (like cheese) I am able to go to the toilet though, not lactose btw. Is there a way to circumvent this issue?
I’m guessing my body cant digest its protein without fat. I am willing to eat more fat by the way I’m just worried about gaining weight because I plan on taking aspirin.
Libido is very high as well, I am also wondering about thag.
-
@CarbCarbCarbs said in Ultra Low fat Diet Logs <20g (mostly saturated):
yes I know nitrates are bad but the vitamin c helps with that
Mind the association nitrates, acid and fat. No lemon juice on smoked salmon.
I never eat an orange just before eating ham.
These reactive nitrogen species, particularly nitrogen dioxide (NO2), can then react with unsaturated fatty acids (UFAs) present in the diet or from cell membranes.
This reaction, known as nitration, can occur through various mechanisms, leading to the formation of NO2-FAs. -
@CarbCarbCarbs I think the dairy is probably very good at forming feces. I was on a mostly skim milk diet for a good few weeks and was able to have solid poops 2-3 times a day.
I think one of the best ways to monitor whether the libido is from estrogen or not is if you are having satisfying orgasms. If you feel a constant need but it doesn't feel good in the end then it's probably estrogen.
-
@LucH so is there a work around this? And what the detrimental effects of NOFAS? I saw this when searching it up:
NO2-FAs have several anti-inflammatory actions, inhibiting neutrophils, platelets and macrophages.
-
This post is deleted! -
@CarbCarbCarbs said in Ultra Low fat Diet Logs <20g (mostly saturated):
NO2-FAs have several anti-inflammatory actions, inhibiting neutrophils, platelets and macrophages.
The combination of these three elements – nitrites + acid + lipids > 10% – leads to the formation of nitrosamines. Note the 10% threshold. If these molecules remain nitrates (salad in winter), no problem if moderate.
Foods rich in nitrates are mainly vegetables, especially leafy greens and beets. Spinach, romaine lettuce, kale, arugula, Swiss chard, beets, and celery are examples of vegetables containing high concentrations of nitrates.
To a certain extent, nitrites are beneficial (coronary dilation), but it’s better to avoid NO2 (Viagra) and favor CO2 ("Protective CO2 and aging" - raypeat.com). Carbon dioxide is increasingly used to prevent inflammation and edema, according to RP.
Note: NO2-FAs have several anti-inflammatory actions, inhibiting neutrophils, platelets and macrophages (NO2-FAs acts by inhibiting the activation of the process).- Platelets:
NO2-FAs can inhibit platelet activation, which is a key component in inflammatory processes. - Macrophages:
NO2-FAs can influence macrophage polarization and activity, potentially shifting them towards an anti-inflammatory phenotype. Must be limited in time.
How to limit the conversion of nitrites into carcinogenic nitrosamines
Key ideas- Nitrites can lead to the formation of carcinogenic nitrosamines.
- Vitamin C, vitamin E, and certain polyphenols (EGCG from green tea) could trap nitrites and inhibit the formation of NAs (N-nitrosamines), under certain conditions, by a range of 60 to 80%, depending on the context.
- The effectiveness of these actions must be qualified: It may be more beneficial to ingest foods that already contain the inhibitors, included in the production process.
- The combination of agents acting in a lipophilic and aqueous environment is desirable. The presence of other antioxidants facilitates the inhibition of the nitrosation of secondary amines (digestion).
- Antioxidants trap nitrites (NO3-) and inhibit the formation of NAs by donating electrons to neutralize the reaction.
To be continued on this link (in French, translator needed, but with English references).
https://mirzoune-ciboulette.forumactif.org/t1983-comment-limiter-la-conversion-des-nitrites-en-nitrosamines-cancerigenes#28949 - Platelets:
-
@LucH Thank you I understand now! I am thinking of dropping the ham and going for chicken and green peas cause I need the thiamine
aspirin should be enough to reverse the damage caused right? -
@CarbCarbCarbs said in Ultra Low fat Diet Logs <20g (mostly saturated):
I’m just worried about gaining weight because I plan on taking aspirin.
If you worried about weight, I'd rather learn how to manage insulin spike, specially at breakfast, since it's going to impact the whole day.
Remember you need fat to assimilate lipovitamins A D E K (+ carotens) and convey polyphenols. I'd target minimum 20 % fat.
Each time there is 2 gr PUFA, like this morning when combining ham (+ 1 tsp Andalusian sauce, a kind of spicy mayonnaise, with canola) after oat flakes (simply crunchy, with honey), I try to get SFA from coconut. I put very mince shredded coconut, with my oat flakes.CCC said:
"aspirin should be enough to reverse the damage caused right?"
=> I won't say it so. Aspirin is well-known to be pro-metabolic and is going to moderate prostaglandins (inflammation from Cox 1 & 2) and TNF-alpha , as well NF-kb.For other readers, see useful info on prostaglandins
Tripping Lightly Down the Prostaglandin Pathways
Written by Mary G. Enig, PhD and Sally Fallon. January 2000http://www.westonaprice.org/know-your-fats/tripping-lightly-down-the-prostaglandin-pathways
-
To avoid problem when metabolizing fats: required nutrients. Here the omega-3 pathway.
Have a look at D-6-D and D-5-D (desaturase enzymes). Delta-5 desaturase is the most limited pathway (when eating high fat).
