I have proof of dozens of guys regrowing from "it's over" levels of bald with high pufa diets and pro-nitric oxide things
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@antiracistracist send this to kevin mann
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Castrated men don't go bald. You know what you gotta do now OP...
https://en.wikipedia.org/wiki/Pattern_hair_lossI was raised on a high pufa standard healthy diet (chicken for protein/low saturated fat/complex carbs/veggies). I was suffering immense hairloss into my twenties and suffer MPB. Where's my success story, op??
This has been explained ad nauseum by Peat and Danny, so I don't blame Danny blocking you. I'll answer once then block you myself for your anti-curiosity, anti-masculine, vigorless copium:
Estrogen is an anti-inflammatory stress hormone: it increases local water retention. This increases local bloodflow and possibly decalcification. It's what happens in the long term -- which is what happened to me, and most others. Many people on mainstream estrogenic protocols experience a reversal of results after a year or more. If you already have an excellent metabolism, I'm sure you can resist a lot of stressors and chronic estrogen application. I don't believe I ever heard Danny or Ray say the estrogenic process doesn't yield hair growth, but that long term they are toxic and don't fix the underlying condition. Chronically elevated estrogen WILL lower metabolism, and with it temperature and bloodflow in various tissues in the body.
In, I believe minoxidil, one of the metabolites is actually functionally like epitestosterone (same as spironolactone) -- something the body wants to convert testosterone to, in hair follicle locations, but what the impaired metabolism blocks. In this case it's not actually the function of estrogen.
Courtesy of Elephanto on RPF:
"An high IGF1/IGFBP-3 ratio is predictive of baldness. Estrogen increases IGF-1 and inhibits IGFBP-3.
Increasing IGF-1 by a single standard deviation increases balding risks by 31%. Increasing IGFBP-3 by a single standard deviation decreases balding risks by 38%.
https://www.mcgill.ca/pollak-lab/fi...ing_plasma_insulin_like_growth_factor_1_0.pdf
Estradiol, progesterone, and transforming growth factor alpha regulate insulin-like growth factor binding protein-3 (IGFBP3) expression in mouse en... - PubMed - NCBI
In these two studies, they find High Estrogen and Cortisol in balding men :
Hormonal basis of male and female androgenic alopecia: clinical relevance. - PubMed - NCBIHormonal parameters in androgenetic hair loss in the male. - PubMed - NCBI
The cascade often starts before Estrogen. Endotoxins and/or Serotonin promote chronically high Cortisol which promotes Estrogen synthesis. Cortisol promotes DHT. DHEA-S which is significantly higher in balding men is a marker of stress. Inflammatory cytokines and prostaglandins also induce aromatase expression and estrogen synthesis.
Serum elevation of dehydroepiandrosterone sulfate associated with male pattern baldness in young men. - PubMed - NCBI
Acute and chronic stress increase DHEAS concentrations in rhesus monkeys
Zinc, which is anti-estrogenic, anti-inflammatory, anti-bacterial, anti-cortisol and inhibits Nitric Oxide is significantly lower in all types of hair loss, including male pattern baldness.
Analysis of Serum Zinc and Copper Concentrations in Hair Loss
http://www.ijdvl.com/article.asp?is...ssue=6;spage=741;epage=741;aulast=Aiempanakit"
Game, Set, OP sacrifices his nuts in a tragic vanity accident, Match.
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Also, explain where and how Hans Amato has left the Peaty paradigm. There has been disagreement within the bioenergetic sphere of how impactful CICO is to sustained weightloss. he practices CICO to yield results. Peat himself supported CICO so this is no problem. That’s the only Significant diversion I know of
