“Long COVID” Suggestions
-
If you subscribe to the idea of “long COVID”, what would your Peaty suggestions be in how to improve and ultimately resolve the condition? Has anyone tried anything or carried out a routine in which they noticed substantial improvements if they felt they were experiencing LC?
-
@peatyRD said in “Long COVID” Suggestions:
If you subscribe to the idea of “long COVID”, what would your Peaty suggestions be in how to improve and ultimately resolve the condition?
Not specially peaty but here is an interesting talk on long convid.
I've more info on prevention and how to kill usual covid, if wanted. Don't let it come and remain too long.
I've had long covid but rather weak and manage to recup well.Excerpt:
Nicotine for long covid
S.Holmes says:
Bioenergetic forum > Case studies
https://bioenergetic.forum/topic/3617/nicotine-for-long-covid
This is a very long video, but packed full of good information.
I've had covid 3 times which ultimately became long covid. My 84 year old mother had developed long covid at the same time. We both used the 7 mg nicotine patches for 7 days and our symptoms improved dramatically. Nicotine sounds like a wonder drug. I decided I'm going to stay on 3.5 mg throughout flu season.
https://rumble.com/v4obxnq-episode-04.10.2024-the-other-n-word.htmlThe_black_jew says:
@S.Holmes
It works but the acetylcholine stim can get overloaded which can cause nausea and a pretty horrific depression.
The human equivalent dose that stimulates nad+ is like 1-2mg over the course of an entire day.S.Holmes says:
Good to know.
Dr. Ardis uses a 3 mg patch every day, and he said he will for the rest of his life. I wonder how he is able to do that without experiencing depression.
Thoughts?The_black_jew:
@S.Holmes
3mg over a full day is probably totally fine.
I’m talking more about the 4mg 3x a day some people do.ThinPicking:
It's conceivable any CNS stimulant would be effective in any fatigue syndrome of any kind.
This is pretty common in urban parts of Britain because life in them is so amusingly miserable. -
"Officially" that's "post-acute sequelae of COVID-19" or "PASC". Anyone contenting with syptoms allegedly associated with it should probably escape both abstractive language traps as soon as possible. The mechanism of the "virus" in the presence and persistence of an energy deficit yields the symptoms. Address the mechanism and the energy deficit.
Fundamentally they're not distinct. Therefore, do and take seriously all bioenergetic things.
-
@LucH your presence here is legendary to me.
-
@ThinPicking said in “Long COVID” Suggestions:
your presence here is legendary to me.
Thanks for encouragement. Not too often, otherwise I could catch a big neck
As I was re-reading my notes to prepare a doc on useful molecules against covid,
I found this one:
Dramatic Decrease of Vitamin K2 Subtype Menaquinone-7 in COVID-19 Patients
doi: 10.3390/antiox11071235 2022
The COVID-19 patients had significantly lower MK7 levels than non-COVID-19 pneumonia patients and healthy controls.
Conclusions: The present data identified significantly decreased vitamin K1, K2 (MK7), and increased MK4 levels in patients with COVID-19 compared to healthy controls. Vitamin K2 (MK7) was lowest in COVID-19 patients irrespective of potentially fatal courses, indicating consumption of this VK subtype by COVID-19 immanent effects, most probably inflammatory and oxidative stress factors.
NB: I take K2 MK4 every day (1 drop 1 000 mcg) but once a week a high level of a mix, included 1200-1500 mcg K1 (1200 mcg) and KM7 (100 mcg)
Life Extension, Super K, 2600 µg,And this one:
Reduced Vitamin K Status as A Potentially Modifiable Prognostic Risk Factor in COVID-19
https://www.preprints.org/manuscript/202004.0457/v1
Vitamin K status was reduced in patients with COVID-19 and related to poor prognosis. Also, low vitamin K status seems to be associated with accelerated elastin degradation. An intervention trial is now needed to assess whether vitamin K administration improves outcome in patients with COVID-19.Why?
Coagulopathy and thromboembolism are prevalent in severe COVID-19 and relate to decreased survival. Coagulation is an intricate balance between clot promoting and dissolving processes in which vitamin K plays a well-known role. We hypothesized that vitamin K status is reduced in patients with severe COVID-19. -
Here is another suggestion to consider.
Randomized, waitlist-controlled trial of
Cordyceps sinensis mycelium culture extract
(Cs4) for long COVID patients in Hong Kong (2025)Highlights
• Cordyceps sinensis mycelium culture extract (Cs4) significantly reduced long COVID symptoms.
• Cs4 improved both physical and mental quality of life.
• Cs4 is a safe option as no severe adverse events were reported
-
@LucH said in “Long COVID” Suggestions:
Thanks for encouragement. Not too often, otherwise I could catch a big neck
Last one. Great attitude.
-
Makes sense to me. A clotting factor seems entirely consistent with a bigger picture of circulatory dysfunction.
Why. Why oh why in 2020. Did we take a disease that seems to boil down to the over expression of ace2, call it covid and try to plug the hole.
-
I didn't have COVID but I had bronchitis. But nevertheless I had symptoms after COVID which I believe would be common to long COVID such as
chronic fatigue symptoms
low RBC bordering on anemia
lower spO2
hardness of breathingedema, expressed in ankles and belly, would be a sign I would watch out for as it would lead to heart failure
in fact, over a 2 year period I've been to the ICU twice and the ER once and each time learned more about it in order to avoid recurrence
I had to learn my own way without having to take the many drugs among which are hypertensive medication involving diuretics which would keep my lungs from being flooded with water from edema
I realized that because of having low spO2 (chronically being in the low 80s for stretches daily) led to many of my red blood cells dying, and over many months, this led to an accumulation of dead red blood cells, too much that my lymphatic system was backed up in clearing them quickly as normally they would when not overwhelmed; I still have an enlarged lymph gland sticking out my left elbow as a cyst to remind me I still have dead rbc's backed up in my lymphs; so the continual slow clearing and release of dead rbc's is the source of free iron and carbon monoxide into my bloodstream, in which the lungs would be heavily affected, as carbon monoxide would impair my breathing as it attaches to hemoglobin; the free iron would also have its negative effects w/r to the creation of oxidative stress
this analysis is however not something a doctor would tell me. this came out of my own way of making sense of Peat's writings on heme oxygenase enzyme and how it converts the even more toxic remnants of dead rbc's into the less toxic forms in free iron and carbon monoxide, which are still toxic in themselves only less
even if I were right in my analysis, I still am experimenting on using different peaty substances and devices, and even substances outside peat's province but consistent with his thinking, but so far I am benefiting from my own therapy as I no longer sleep poorly and no longer suffer from fatigue, no longer have edema from having normal spO2 levels but still have white phlegm from my throat which indicates strong immune system activity in my lungs that thankfully is being cleared out instead of blocking my breathing as thick mucus
I am however not out of the woods yet as I monitor my lung and heart health using an app on my old Samsung Galaxy S phone which has heart rate and oxygen saturation sensors. I find that on the times that correspond to the lung and liver and gallbladder on the TCM organ chart, my charts don't look good still and I'm still a work in progress.