@winters2 said in vitamin E complex?:
Didn’t Ray have a negative attitude towards tocotrienols?
Yes, but it's a question of dosage and frequency, for me. Still useful, but I won't take much of it, if not in cure, thus for a limited period. Ray said T3 has effects on animal liver (enlargement, probably toxic). T3 is however more fluid / mobile, so that it could easily reach its target (inside membrane vessel, for protection; personal deduction). But I conceive T3 in a rather low fraction, in association with alpha and gamma T1. However there are very few studies analyzing a combo of T1 and T3. And not just under12 weeks as it won't probably prove anything (...)
I cite, from "Vitamin E: Estrogen antagonist, energy promoter, and anti-inflammatory"
Besides antagonizing some of the end effects of the toxic fatty acids, vitamin E inhibits lipolysis, lowering the concentration of free fatty acids (the opposite of estrogen’s effect), and it also binds to, and inactivates, free fatty acids. The long saturated carbon chain is very important for its full functioning, and this saturated chain might allow it to serve as a substitute for the omega -9 fats, from which the Mead acid is formed. The unsaturated tocotrienols have hardly been tested for the spectrum of true vitamin E activity, and animal studies have suggested that it may be toxic, since it caused liver enlargement.
One possibly crucial protective effect of vitamin E against the polyunsaturated fatty acids that hasn't been explored is the direct destruction of linolenic and linoleic acid. It is known that bacterial vitamin E is involved in the saturation of unsaturated fatty acids, and it is also known that intestinal bacteria turn linoleic and linolenic acids into the fully saturated stearic acid.