Lobotomize-me athletic logs
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@pittybitty my guy ive been mega dosing aspirin milk with glucose salt taurine glycine for a while now so i dont believe it is stress hormones getting lowered. before peat doing extremely strict keto i was sick every 3 or 4 months so i am probably more prone to getting ill when stress hormones dominate
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i drink a diet manly out of 1.5% fat milk, orange juice glucose salt and supplements
(Mega doses aspirin , vit b2, b1, b3, b7 pregnenalone and dhea, 100 mcg t3, vit A 5k, taurine 4 g, glycine 10g, creatine 6g, succinic acid 4 g seperated doses of 1 g
/////// daily ) (occasional magnesium)is there anything i am missing or doing wrong which hinders my ability to lose weight ?
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@lobotomize youre missing out on 5% milk
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@polba so me drinking 5 liter milk a day and getting 75g fat isnt enough? I need 250 g fat?
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So affer switching to 0 fat milk 2 things happend
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Started losing massive amounts of weight
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I became ammonia toxic heres why
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Gastric Buffering and Proteolytic Failure
Consuming ~10 liters of milk per day created a massive alkaline buffer that neutralized gastric hydrochloric acid (HCl). With stomach pH remaining too high, the enzyme pepsin stayed inactive. As a result, milk protein (casein) failed to clot or denature and instead entered the lower gastrointestinal tract as an undigested proteolytic sludge. -
Bacterial Putrefaction and Ammonia Generation
This undigested protein became a large feedstock for proteolytic bacteria in the colon. Through putrefaction, these bacteria stripped nitrogen from amino acids, generating large amounts of ammonia (NH₃) directly within the gut. -
The pH Trap and Non-Ionic Diffusion
In a healthy, slightly acidic gut, ammonia (NH₃) is protonated into ammonium (NH₄⁺). Because NH₄⁺ is charged and water-soluble, it is trapped in feces and excreted. However, the alkaline environment maintained by extreme milk intake prevented this conversion, leaving nitrogen predominantly as NH₃ gas. Through non-ionic diffusion, this lipid-soluble gas crossed the gut lining, entered the portal vein, and overwhelmed hepatic clearance. -
Metabolic ATP Bankruptcy
Once ammonia reached systemic circulation, it imposed a severe metabolic burden. The liver requires four ATP molecules to convert one molecule of ammonia into urea. Concurrent use of aspirin (a mitochondrial uncoupler) and T3 meant baseline cellular energy was already compromised. Hepatic detoxification diverted available ATP toward ammonia clearance, leaving insufficient energy for skeletal muscle function.
Krebs cycle inhibition: Enzymatic disruption in chest and triceps muscle limited ATP production, eliminating the explosive power required for a bench press.
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@lobotomize said in Lobotomize-me athletic logs:
So affer switching to 0 fat milk 2 things happend
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Started losing massive amounts of weight
-
I became ammonia toxic heres why
-
Gastric Buffering and Proteolytic Failure
Consuming ~10 liters of milk per day created a massive alkaline buffer that neutralized gastric hydrochloric acid (HCl). With stomach pH remaining too high, the enzyme pepsin stayed inactive. As a result, milk protein (casein) failed to clot or denature and instead entered the lower gastrointestinal tract as an undigested proteolytic sludge. -
Bacterial Putrefaction and Ammonia Generation
This undigested protein became a large feedstock for proteolytic bacteria in the colon. Through putrefaction, these bacteria stripped nitrogen from amino acids, generating large amounts of ammonia (NH₃) directly within the gut. -
The pH Trap and Non-Ionic Diffusion
In a healthy, slightly acidic gut, ammonia (NH₃) is protonated into ammonium (NH₄⁺). Because NH₄⁺ is charged and water-soluble, it is trapped in feces and excreted. However, the alkaline environment maintained by extreme milk intake prevented this conversion, leaving nitrogen predominantly as NH₃ gas. Through non-ionic diffusion, this lipid-soluble gas crossed the gut lining, entered the portal vein, and overwhelmed hepatic clearance. -
Metabolic ATP Bankruptcy
Once ammonia reached systemic circulation, it imposed a severe metabolic burden. The liver requires four ATP molecules to convert one molecule of ammonia into urea. Concurrent use of aspirin (a mitochondrial uncoupler) and T3 meant baseline cellular energy was already compromised. Hepatic detoxification diverted available ATP toward ammonia clearance, leaving insufficient energy for skeletal muscle function.
Krebs cycle inhibition: Enzymatic disruption in chest and triceps muscle limited ATP production, eliminating the explosive power required for a bench press.
How do you know that is the cause of your symptoms from no fat milk and/or amonia toxicity?
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@user1 which symptoms?
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@lobotomize said in Lobotomize-me athletic logs:
@user1 which symptoms?
how do you know you got ammonia toxicity?
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@user1 Feel like my muscles are "poisoned" or "heavy," especially during the second set or compound lifts.
Circadian rhythm delay.
Extreme grogginess in the mornings.
Milk being the only thing I changed that could have contributed to these
