Help I can't poop.
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I went on antibiotics and now I can't poop, I had to take laxatives just so I could defecate.
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@monkey11 after going on antibiotics once after I had never taken them for over 20 years I read a paper from another country, I think it was German that had tables for the different types of probiotics in different foods. Its conclusion was that repopulating good bacteria with as many varied strains as possible was helpful after taking antibiotics. I had really good BMS before and then they were affected after antibiotics so I ate a little bit of fermented foods everyday from sauerkraut, buttermilk, yogurt, kefir, kombucha, miso. Not tons but a serving and I also took saccharomyces boulardi Jarrow brand. I think I did it for no more than a month It made everything perfect again. I also put my cat on s.boulardi after antibiotics and it helped her.
I had been following peats work for years before all this too and I know peat people steer clear of fermented foods but I went with the paper I read for my situation over how people in general interpret peats work. Glad I did too.
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Carrot salad, often. Pau D'arco tea, and cascara sagrada should create the need to have a bowel movement in a normal situation.
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Right with what has been said.
But you need to optimize the context:- 30 gr fibers, functional MMC and gut motility.
MMC= Motor Migrating Complex. I can develop / explain if wanted. - Some useful nutrients for the communication between the brain and the intestines, through the vagus nerve.
Thiamine as cofactor for autonomic nervous system
When cells lack thiamine (B1) the lower regions of the brain – responsible for the coordination of the autonomic nervous system – there are metabolic derangements in the brain susceptible to produce dysfunctional autonomic outputs and misfiring, which goes on to exert detrimental effects on every bodily system – including the gastrointestinal organs.
Thiamine is one of them. - Some useful nutrients to feed gut bacteria. Make a search with “Biosynthesis of Vitamins by Probiotic Bacteria”. Or I can give you a link.
Hope it can help.
- 30 gr fibers, functional MMC and gut motility.
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It's possible you lowered serotonin too much with the antibiotics. Constipation is a sign of low serotonin. Maybe try some oatbran or carrot salad.
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True enough what you said, as this applies to most people. And I believe most people to be magnesium a deficient, and as such needs serotonin to drive bowel movement.
But with a food lifestyle that supplies one with adequate magnesium intake, bowel movement is not dependent on having a gut flora teeming with microbes that supply serotonin to drive bowel movement.
I went from being dependent on serotonin to being dependent on magnesium to drive bowel movement, and it has worked well for me.
Lately, I was prevailed upon to take a calcium channel blocker (Amlodipine) for my high blood pressure. One of the side effects is constipation, and I experienced more difficulty with my bowel movement. This is not surprising, as with the movement of calcium into and out of the cell altered , there is a change in the strong gradient between internal and external calcium ( external calcium being around 15000 more than internal calcium) which drives muscle contraction and relaxation. This would affect peristalsis, involving the gut muscles that drive bowel movement. The energy from the magnesium-ATP complex is somehow affected negatively by the calcium channel blockers, and this makes the bowel movement less natural and less effortless.
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Agree with @yerrag, Magnesium is often key with hard stools, especially when it's the chronic dark, hard and dry, even though i don't think that is the OP's problem in this case.
Full doses of milk of magnesia or "USP" grade epsom salt (Mag-Sulfate) as per package instructions. Or if you don't want that sudden effect you can microdose these several times per day with 1/4 or 1/5th doses. Other Magnesiums would work but their labels won't contain instructions on how to deal with constipation ... you'd have to look that up yourself or experiment.
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@monkey11 You don't necessarily need to take anything for it. Certain yoga exercises absolutely prevent constipation.
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I have no idea how PTH would influence magnesium. It may but in away where magnesium would be indirectly affected in the sense where CCBs would have a systemic influence where all are affected.
I can speak to my personal observation. I regularly monitor my acid-base balance where I test the pH of both my urine and saliva.
Despite my high blood pressure, when I am not taking bp meds, I see more of a state of balance where my body is given free reign to adapt optimally as it area fit to a given pathological influence, which in my case involves a recurring unresolved state of low-grade infection and exposure to lead toxin. One aspect is in the way my body strives to achieve a state of acid base balance.
I don't feel any arthritic pains in my joints. I don't have a problem with my bladder holding on to urine. However, I wake up often to pee at night, which I associate with water being produced as a product of redox reactions where antioxidants neutralize spillover ROS, which comes to my immune system creating ROS to try overcoming pathogenic microbes and toxins. Still, I look for ways to resolve the infection and toxic state in order to resolve the root causes. While the root causes remain unresolved, my body is remarkable in adapting to this unresolved state. A symptom of this adaptation to the unresolved state is hypertension.
When taking the calcium channel blocker, I see lower bp but I also feel the price I have to pay for the appearance of health in the form of lower blood pressure.
My urine and saliva pH testing would show a marked departure from my state of acid-base balance. The CCB interfering with the calcium channel is expressed clearly when my urine and saliva pH testing reveals a constant imbalance in acid base regulation. Unlike when I am not taking CCB's, my body is always unable to catch up in ridding itself of excess acidity during rest, which occurs at night. So, I would urinate more frequently during both day and night, and I would be peeing in my pants (and feeling old and incontinent) even at times when my bladder is not even half-full. My knees and ankles would feel sore as well, which I suspect is the effect of uric acid precipitating on my joints.
If not for taking furosemide and spironolactone, both being diuretics, I would be developing edema at my ankles and my waistline would bloat from ascites developing.
I should stop now, as the side effects from taking bp medication (so far I have mentioned amlodipine, furosemide, and spironolactone) can have me go on endlessly.
And I am going off topic somehow, but I only want to bring home the point that interfering with the calcium channel has systemic effects. It just isn't worth it to take calcium channel blockers to appear normal bp-wise while ignoring the effects on other areas which our doctors trivialize and even consider coincidental.
I tried to find some authoritative sources on the effects of CCB's, but I have difficulty doing that. My research skills are not as good as that of you and @Amazoniac 's, and what I find are mainly topics that are peripheral and not central to the issue. I suspect it is so because such research or inquiries are frowned upon by the establishment. Unfortunately, I have been spoiled by Peat doing the legwork for me, and I wished he had expounded in his writings on CCB's.
One thing one of his last newsletters mentioned was that when the body is doing osteoclastic activity, it cannot at the same time do osteoblastic activity. Osteoclasts break down bone to provide the body with calcium when the PTH signal is high. In this situation, no osteoblastic activity can be done. Bone building requires CO2, so it goes that during this time mitochondrial respiration is halted to give way to other metabolic pathways. These other metabolic pathways often involve producing acids, such as lactic acids and keto acids.
When the metabolic pathway becomes anything other than mitochondrial respiration, the body becomes acidic more and more. And all sorts of imbalances and pathology result, especially when it becomes a permanent and chronic feature.
Even when one has adequate mineral stores in the body such as potassium and magnesium, they are not utilized in the way they should be, because the general condition that permeates, as determined by an acid-base imbalance,
overrules the salutary nature of these substances. -
@yerrag How high of a BP are we talking about? I remember Ray saying high blood pressure as we age is an adaptive protective mechanism. Although I assume this has limits. I guess it would greatly depend on your age as well.
Have you tried ACE inhibitors instead of CCBs? The swelling sounds more like a RAAS aldosterone issue to me. Most ACE inhibitors are a little "softer" on the side-effect spectrum as well. A high-potassium diet can also help flush excessive fluids out. I say "high," but in reality just shooting for the RDA of 4 grams will likely be a big aid. You'd be surprised just how many people fall short of K's RDA on a daily basis.
As for the PTH-magnesium thing the only reason I mentioned that was based on the limited data I've seen on CCB's they seem to stimulate PTH. Or some of them do at least (the dihydropyridine class). And since magnesium helps modulate PTH similar to how dietary calcium does, you may be responding well to magnesium because it's helping to slow down the extracellular Ca concentration.
Not to get too far off topic myself, but this could have possibly been set in motion by unresolved hypothyroidism, which has been shown to decrease calcium excretion via renal impairment in humans. Once on thyroid replacement the problem should resolve itself, or at least get substantially better.
But in the event kidney's have reduced ability to get rid of calcium, and CCB's increase the extracellular Ca from PTH stimulation, you might be left in precarious situation where you actually feel better not taking the CCBs despite higher BP. And it would explain why you respond well to magnesium too, as it's ability to modulate PTH independent of calcium networks.
Lots of speculation though, would be nice to see some tests specifically measuring the TSH, PTH, 1,25-D, and calcium for a better understanding. If any (or all) are elevated it would be a little more clear of what's happening.
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My pooping mixture:
1 teaspoon fine baking soda (arm n hammer)
2 TBsp magnesium bisglycinate
2 teaspoons instant coffee
250 ml skim milk
pinch of saltrepeat if doesn't work
POOP TIME
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@Mulloch94 said in Help I can't poop.:
How high of a BP are we talking about? I remember Ray saying high blood pressure as we age is an adaptive protective mechanism. Although I assume this has limits. I guess it would greatly depend on your age as well.
Yes, it is an adaptive mechanism. And it is age-related but only insofar as with age one deals with the cumulative effects of a lifetime of unresolved chronic stresses that range from toxins, infections, and physical and even mental (emotional, psychological, and even spiritual) trauma.
So I have gone all the way to 240/160 and now I have been able to lower it to 185/120, without prescription meds. Through it all, no headaches. No nothing. When I had heart failure last year, it was due to a self-induced bronchitis that deprived me of a stable supply of oxygen that spiraled into heart failure, which now I am fully recovered from. Saying this as this is what my doctor and friends would tell me- that it was my high blood pressure. But I don't have to explain further as that is how lesser minds (I mean this) work as they can't separate wheat from the chaff.
Anyway, to make this short, the root causes I have already identified but working on removing these root causes requires more effort and I am halfway there as seen in the lowered bp.
I monitor many metrics at home, which is not something this forum (except a soul or two) is interested in, but suffice it to say that.these metrics confirm my better health when I'm not taking the prescription drugs.
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@Mulloch94 said in Help I can't poop.:
Have you tried ACE inhibitors instead of CCBs? The swelling sounds more like a RAAS aldosterone issue to me. Most ACE inhibitors are a little "softer" on the side-effect spectrum as well. A high-potassium diet can also help flush excessive fluids out. I say "high," but in reality just shooting for the RDA of 4 grams will likely be a big aid. You'd be surprised just how many people fall short of K's RDA on a daily basis.
I don't intend to try ACE inhibitors or ARBs or anything else in the AMA pharmacopeia. All are David Copperfield's masking as Jesus. All have side effects, as clearly just the CCB is producing edema and the doc would prescribe diuretics along with the CCB but he can't fool me. I know the Furosemide and Spironolactone is not to lower bp but to mitigate the edematous effect of CCBs. And then I would have cramps, and that would be the side effect of diuretics and that would be why they gibe me potassium tablets. And the doc would also prescribe statins as well as a blood sugar drug and a nitrate for angina. I never took the statins and the blood sugar drug as I don't have a problem needing those things. When I was taking the nitrate for angina, my plethysmographs (on my samsung s10) look worse than without them.
Just because Ray Peat likes some drugs such as ARBs does not mean they are good. Ray likes to tell us to take carbon anhydrase inhibitors to ensure more availability of CO2 but he never could convince me to as why doesn't he press his message to improve mitochondrial respiration to just simply produce more CO2 (in effect he considers it wasted effort as most people are too impatient to go about improving their metabolism- IMHO people are just used to taking drugs that resolve issues in an instant. Most people even in the bioenergetics community don't see magnesium, for example, as something whose stores you have to build for a year to really improve bowel movement, and just take magnesium expecting an instant resolution for constipation).
On potassium, I hear you. We're on the same level understanding the importance of having adequate potassium stores.
Potassium is a tricky subject. When ournblood potassium is high, doctors tell us to stop eating foods rich in potassium. It's mind-blowingly stupid or unthoughtful of them to mislead us to think that. Serum potassium is high because the ecf is very acidic, and the cellular potassium lining the membranes have to leave the membranes to exchange for hydronium ions in the blood - in an effort to lower the highly acidic ECF. Why is this blood acidic? One reason is that the CCB is causing this acidity. But this is something people don't know because they never are interested in testing their urine and saliva. I've proven this by sharing how this is done in RPF, and no one is interested. They love only to hear from gurus and not from an interloper whose ideas make sense but have no designer label.
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@Mulloch94 said in Help I can't poop.:
Notto get too far off topic myself, but this could have possibly been set in motion by unresolved hypothyroidism, which has been shown to decrease calcium excretion via renal impairment in humans. Once on thyroid replacement the problem shou
I haven't been hypothyroid since 2017. I check my QTc (a metric used in ECGs as a proxy for hypothyroidism in measuring heart muscle relaxation time) daily and it isn't hypothyroid. also, my temperature is good. Not only that, my stable and normal blood sugar is confirmed by OGTT )Oral glucose tolerance test) curves which I use in place of the highly imprecise and irrelevant HbA1c all people here use (in deference to the medical tyranny of AMA) and my acid-base balance is optimal. Having good blood sugar and acid-base balance is, in my humble opinion, a hallmark of a very good mitochondrial metabolism.
I have taken care of the basics so as to have a firmer base to tackle my issue with hypertension. Without which I would always be secondguessing what is causing my high bp.
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@Mulloch94 said in Help I can't poop.:
But in the event kidney's have reduced ability to get rid of calcium, and CCB's increase the extracellular Ca from PTH stimulation, you might be left in precarious situation where you actually feel better not taking the CCBs despite higher BP. And it would explain why you respond well to magnesium too, as it's ability to modulate PTH independent of calcium networks.
The kidney is the least of my concern. I have posted many times in RPF on why the use of serum creatinine as a basis for determining kidney health is bogus. I believe it's one of the biggest lies or myths in medicine. It's because I use the creatinine clearance test based on collecting urine for 24 hours to help determine the glomerular filtration rate. The results are better than something that really is an estimate (the e in eGFR clearly means that) based on age, sex, race - which I consider highly unusable in a setting where science means anything.
I believe that the main cause of kidney dysfunction is simply poor acid-base balance. This leads to internal calcification, towards the development of fibrosis. If we just know how to keep our acid-base balance, many issues that vex us would be a non-issue. Kidney dysfunction is one of them. Cancer another.
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@yerrag
Lowering a raised blood pressure does absolutely nothing, unless it is exceptionally high. All modern BP medication have a high incidence of stroke and heart attacks, plus a lot of other nasty side effects. The ALLHAT study found that amlodipine is no more effective than water pills in preventing deaths. Fortunately, there are easy safe ways to dramatically lower BP without drugs. Here is an article that I wrote that you may find of benefit: -
@Dr-John-H Thank you very much for writing and sharing that article with me. It validates in many ways my approach towards dealing with my hypertension, and gives me additional insights in continuing my efforts to lower blood pressure naturally.
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@yerrag Your Welcome!
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@yerrag Yeah that's a pretty serious BP at 240/160. Definitely not the "slightly elevated" adaptive BP of old age. Are you sure you've eliminated any possible hypothyroidism? Checking the QT interval is very hit and miss, simply because there's so many things that can distort that (not just hypothyroidism). And the symptom itself may not show in everyone who is hypothyroid. What about your blood lactate levels? Which will indicate a metabolic dysfunction if elevated. In my experience hypothyroidism, or TSH (more specifically TRH), is a big cause of hypertension. My BP wasn't the greatest until I got on thyroid replacement. TRH can act a lot like adrenaline, pushing the heart rate up while inducing vasoconstriction. Of course all sort of downstream abnormalities will manifest themselves, like altered calcium metabolism. And why magnesium might help this (albeit only acutely until thyroid is corrected).
