Posts made by dht

@alfredoolivas said in Pro Metabolic Substances Tier List:

Haidut was ahead of the time, he made these posts nearly 10 years ago, long before anyone was talking about it.

Indeed

@alfredoolivas said in Pro Metabolic Substances Tier List:

The issue is that it is hard to get DHT nowadays, China has had a major crackdown on steroids, and all steroids are more scarce. DHT has basically disappeared and but PPL customer service said they plan to restock it.

There will always be labs in China and Hong Kong to synthesize drugs, banning testosterone raw materials is not real it’s like banning bitcoin

@sushi_is_cringe im sure dht is suppressive at supraphysiological doses that's why I plan to continue supplementing the upstream hormones rather then continuing to use test esters

@sushi_is_cringe i inject it,

i don't believe dht is suppressive at 25mgs because higher estrogen levels cause the hypothalamus to reduce the secretion of GnRh, which in turn means suppressed LH and FSH production from the pituitary. so before the exogenous test (or aromatizable androgen) even has a chance to cause suppression through androgen receptor mediated feedback, you are already suppressed from the aromatization.

@sushi_is_cringe currently taking 25mgs every day, what does roa mean?

https://x.com/BowTiedUM/status/1894894131886919965

i tried DHT enanthate solo for a week and felt good with no signs of suppression and then added test prop in and it made the experience worse probably due to low pregnenolone production.

@alfredoolivas Interesting, I saw Ben Westgate on twitter say that testosterone over 1000mgs per week is non suppressive because the aromatase enzyme caps out at 600mgs per week so the excess Testosterone and DHT acts as AI to bring back LH and FSH signaling the same way pregnenolone does at high doses whilst shutdown

@alfredoolivas what about shortening your life? RBC production? do you donate blood? supplement iron? at that point why just not take the harmless precursors with progesterone and thyroid as an AI?

@alfredoolivas still though, 2 grams? Speaking of PUFA depletion I've noticed since starting DHT I have required less thyroid for symptom relief

@alfredoolivas aromatizable androgen esters are poison im ngl, i feel considerably better on pregnenolone, dhea and progesterone compared to test E and P at 6-7mgs per day with DHT Enanthate, so i have no idea how you tolerate 2 grams per week

@alfredoolivas said in Pro Metabolic Substances Tier List:

But maybe I should buy heavy metal laden DNP...

worrying about heavy metals in 200mgs of DNP when you claim to have taken 2100mgs per week of testosterone for 9 months is kind of hilarious

@Crypt-Keeper purple panda labs if you want raws
or potentially this
https://healthnatura.com/estrogen-oil-1-oz/

@alfredoolivas I wasn't advocating for the use of DHT without an estrogen precursor. The few people hyping up DHT Enanthate on twitter are using exogenous T and E2 alongside DHT.

https://x.com/BenWestgate_/status/1838407090739589474

https://x.com/solothesensei/status/1878763267038105872

https://x.com/BowTiedUM/status/1886096759832641778

Whether if it is just "hype" or not, estered hormones are going to out perform non estered hormones in its effects every single time.

I don't think it is that simple. The rapid peaks of base DHT saturates 3a-HSD and 3b-HSD enzymes which leads to immediate conversion to the inactive metabolites. DHT Enanthates esterification leads to a slower release into the bloodstream, reducing the peak concentration, prolonging the duration and increasing free DHT levels. This slower release means 3a-HSD and 3b-HSD enzymes are not overwhelmed with a large bolus of DHT at once, which would rapidly convert into the inactive metabolites. Instead the steady release allows DHT to remain active longer, as the enzymes are not as quickly saturated, leading to a higher free hormone level and a greater androgenic effect on tissues compared to the base form. Essentially the ester out competes the inactivation of DHT which is probably why these body re compositions are not being achieved on high doses of base DHT dissolved in either DMSO or Tocopherol.

@sushi_is_cringe nope just somebody very interested in DHT.

Despite the limitations of base DHT and its duration of action on tissues, I have personally seen results from using it. Over weeks of experimentation I've experienced crashed estrogen, increased magnetism, measurable penis growth and reversal of some pre-existing gyno. However achieving the physical results required applying high doses topically and ingesting even higher doses. I believe including an estrogen precursor like Testosterone or DHEA at ratios to be experimented with, would help even if the user was on DHT Enanthate with crashed e2.

@Crypt-Keeper I don't think it is that simple. The rapid peaks of base DHT saturates 3a-HSD and 3b-HSD enzymes which leads to immediate conversion to the inactive metabolites. DHT Enanthates esterification leads to a slower release into the bloodstream, reducing the peak concentration, prolonging the duration and increasing free DHT levels. This slower release means 3a-HSD and 3b-HSD enzymes are not overwhelmed with a large bolus of DHT at once, which would rapidly convert into the inactive metabolites. Instead the steady release allows DHT to remain active longer, as the enzymes are not as quickly saturated, leading to a higher free hormone level and a greater androgenic effect on tissues compared to the base form. Essentially the ester out competes the inactivation of DHT which is probably why these body re compositions are not being achieved on high doses of base DHT dissolved in either DMSO or Tocopherol.

@flux In my experience exogenous t4 is non suppressive to TSH and stopping t3 for at least 4 days before a blood test has my TSH normalized at around 2.

@jamezb46 I successfully dissolved 2250mg of DHT in 30 milliliters of d alpha-tocopherol (at 1,000 IU/mL). However, I added around 8 mL of olive oil for its oleic acid content which is necessary for chylomicron transport of the hormone. This brought the concentration from 75mg/mL to 65mg/mL. Based on Ray's patent and his 3.5:9 ratio of fat to alpha-tocopherol, I speculate the ratio (in mg not mL) of hormone to alpha-tocopherol to oleic acid could range from 1:9:1 to 1:9:5. Finding the least amount of oleic acid required is the next thing I am going to test through trial and error.