Lectins, saponins and gliadin increase intestinal permeability and allow increased passage of gut bacteria from intestinal lumen to peripheral circulation.
LPS - an antigen found in gram-negative bacteria cell membranes - interacts with TLR-4, leading to inflammatory cytokine production and low-grade chronic inflammation, which is at the root of insulin resistance. Insulin resistance is recognized to induce the metabolic syndrome, including T2D. Endotoxemia-induced insulin resistance in T2D patients may be exacerbated, in part, by antinutrients.
Posts made by LucH
-
RE: Share your PUFA story
-
RE: Share your PUFA story
@psi said in Share your PUFA story:
it could have been additives in bread.If you eat bread or flakes at breakfast, rice and pasta at the two other meals and perhaps a cookie at midday, don't bother about PUFA. Of course your liver could be sluggish and your glycemia out or the way. Or your thyroid in the cabbages, as we say here.
But have you ever heard of prolamines (gluten) and the impact on your border bross of the intestines (leaky gut due to zonuline action). When you don't pull too much on the rope, you recover at night. we repair via homeostasis, provided you've done it on a moderate way. Not 3 times. -
RE: Macadamia nuts fats okay?
Macadamia nuts
Each nuts is supposed to weight 3 gr.
For 100 gr
Ratio w6/w3 6.29. Ratio Oxalate / Calcium 2.02 (low)
Ratio P / Ca 188 / 85 => 2.21
Note Phosphorus should be between 1 and 2.2. So, it’s Ok. The main problem with nuts, in general, should be oxalates and omega-6.
When I eat macadamia is turns around 5 nuts (x 3 gr). A portion is 30 gr.
For 30 gr you’ve got: 0.39 gr omega-6. OK for me. -
RE: Where do I begin?
@psi said in Where do I begin?:
are you taking coconut oil to purge the bacteria?
Cococonut is also virucide. So, it would be advisable not to take it at the beginning of the process when trying to alleviate the load for liver. No problem with coconut oil if you manage well.
By the way, the way out is easier with MUFA (olive oil or avocado), and still easier with PUFA (white fish and seafood). Need to be known when the liver is overburdened. -
RE: Where do I begin?
@rosy-fingered said in Where do I begin?:
Please share some of your recommended reading material
Hi,
Here is a starting point. Some points could asked a development when you search for details or other sources to justify / explain one point out of current standpoint.
*) “How to Heal Your Metabolism - Stop Blaming Aging for your Slowing Metabolism!”
Kate Deering .
This book is based on the work of Dr. Ray Peat, Dr. Gilbert Ling, Dr Albert Szent-Györgyi, Dr. Broda Barnes, Dr Constance Martin, Dr. Hans Selfe, Dr. Uffe Ravnskov, Dr Lita Lee, Chris Masterjohn, and Josh Rubin.
"How to cure your metabolism" will give you a completely different perspective on what food, supplements and exercise are really. Are they "healthy" for you, when and what intensity, and what are those who can harm you.
This book is based on Kate Deering's experience. Kate Deering is a physiologist (physical exercise), a personal trainer, a nutrition coach and for lifestyle, but is not a doctor.
Editor's note (LucH): it is reliable. Even if we do not have to agree on everything ... We must also put questions about our context ... As already specified, she is a fan of Ray Peat (RP), reread and confirmed by RP, but she has a "practical" approach, given her profile. It is therefore a very good book for the reader who needs a concrete scheme, a framework. If I caricature, it transposes the foundations / the theory defended by Ray Peat, and some others, in a practical approach. She puts the points on the I, with her sensitivity (coaching experience and her life-experience) and draws attention to the context. It is an interesting book to get to know the basics of body physiology and connection with food, in a given context (hypo or normal).
Note: I’ve a summary on my forum (In French; translator needed)
http://mirzoune-ciboulette.forumactif.org/t1347-comment-guerir-votre-metabolisme#15017
Comment guérir votre métabolisme -
RE: Starch is truly slave food
Shifting from FFA oxidation to glucose oxidation
RP has mentioned (same source):
Sugar and thyroid hormone (T3, triiodothyronine) correct many parts of the problem. The conversion of T4 into the active T3 requires glucose, and in diabetes, cells are deprived of glucose. Logically, all diabetics would be functionally hypothyroid. Providing T3 and sugar tends to shift energy metabolism away from the oxidation of fats, back to the oxidation of sugar.
Niacinamide, used in moderate doses, can safely help to restrain the excessive production of free fatty acids, and also helps to limit the wasteful conversion of glucose into fat. There is evidence that diabetics are chronically deficient in niacin. Excess fatty acids in the blood probably divert tryptophan from niacin synthesis into serotonin synthesis.
Note LucH: Niacinamide, not niacin. Moderate dose = maxi 20 - 25 mg B3.
Sodium, which is lost in hypothyroidism and diabetes, increases cellular energy. Diuretics, that cause loss of sodium, can cause apparent diabetes, with increased glucose and fats in the blood. Thyroid, sodium, and glucose work very closely together to maintain cellular energy and stability. -
RE: Starch is truly slave food
@Milk-Destroyer said in Starch is truly slave food:
All starchy foods have a tendency for me to cause blood sugar swings. I do not experience this with non-starchy sugar sources.
Starch causes higher insulin spikes than sucrose (di-saccharide).
=> Dress your carbs when eating potatoes.
How to deal with sugars, before stating which kind you can manage:
We should have taken into account the presence or not, of fibbers, the percentage of fructose to glucose, and the way the sugars are dressed / accompanied, etc.
Ray PEAT (RP) has mentioned
If the stored fats happen to be polyunsaturated, they damage the blood vessels and the mitochondria, suppress thyroid function, and cause “glycation” of proteins. They also damage the pancreas, and impair insulin secretion.
A repeated small stress, or overstimulation of insulin secretion, gradually tends to become amplified by the effects of [an excess of] tryptophan and the polyunsaturated fatty acids, with these fats increasing the formation of serotonin, and serotonin increasing the liberation of the fats.Lowering free fatty acids can restore glucose oxidation
Source:
Glycemia, starch, and sugar in context Ray Peat
http://raypeat.com/articles/articles/glycemia.shtml -
RE: Kumerrow and trans fats
Trans fat comes from altered MUFA and easily from PUFA.
100 % hydrogenated oil won't be a problem as it would be associated with SFA. No double link then. It would handle as a saturated fat. It is supposed to be recognized by our our enzymes.
How is another problem.
Our metabolism can handle max 2% of trans fats. Otherwise it's going to overload the system and create problems: unuseful chains. -
RE: Glucose metabolism - Free Fatty Acids
@Ilias-Ant said in Glucose metabolism - Free Fatty Acids:
actually now i m in low fat high carb diet.... but the most important thing for me is low energy.... that's why i m trying to lift weights etc for workout... i also grounding and I'm out in the sun (shade) for about 2.5-3 hours per day...
Sun helps a lot i can feel the difference...Fine.
As long you consume minimum 20 % fat and optimal protein ratio to weight, it's OK.
For fats:
45 % SFA (coconut, butter, meat preferable free range)
45 % MUFA: e.g. avocado, olive oil (only for veggies), free range poultry, duck, etc.
10 % PUFA, but no need to target 10 gr every day. I target 1.5 w3 et 6 gr w6. original, not processed.
See Kate Deering for useful info on energy metabolism. -
RE: Glucose metabolism - Free Fatty Acids
Hi,
there are several points.
In short:- Dress your carbs (See Jessie Inaupsché for details).
- Get informed on insulin index (II and GI) but don't trust the whole thing before reading RP:
Glycemia, starch, and sugar in context. Ray Peat
http://raypeat.com/articles/articles/glycemia.shtml
How to heal your metabolism. Kate DEERING (book)
https://mirzoune-ciboulette.forumactif.org/t1347-comment-guerir-votre-metabolisme-livre?highlight=gluose+metabolism - If you have a fatty liver (MBI > 25), it will be more difficult to manage insulin spike.
- Make sure to get enough potassium (fruits and vegetables).
- Go for a walk during 15-20' after eating carbs, specially after the evening meal, not to make fat.
- Keep unstressed (yoga or sophrology) to help to metabolize well.
- Pampering your thyroid with optimal nutrients would be fine too.
=> lack of magnesium, iodine. - Later, get informed on the ideal ratio between sodium and potassium. Not now.
-
RE: Starch is truly slave food
@Ecstatic_Hamster said in Starch is truly slave food:
starch is far, far better for us, than sugar. Most of us will get fat and have blood sugar problems on sugar, but well cooked starch, WITH a little fat, works very well.
Everyone won't agree on this subject.
RP found both OK as long as the quality is present and you listen to your body. I can develop this last point if desired.
Shortly said: starch is good as long you go easily to stools twice a day, to avoid stagnation in the second part of the colon. otherwise, dysbiosis will come soon.
Of course you manage well with counter-arguments: oxalates or arsenic in some cases.
Yes, dress your carbs and it will be fine, provided you have no problem with digestive enzymes.
For fruits, you avoid most of the time seeds and vary (...).
For vegetables, you have to manage well legumes (anti-proteinase) and you take into account the specifities: like sulfur for kale or other cruciferous, or oxalates / lectines.Vary and adapt oneself to the situation / to the tolerances.
And don't tell me you have problem with insulin if you drink OJ or eat yoghurt, whenever you do it in the wrong way. I don't drink 20 cl OJ after a pancake with syrup or 200 gr Greek yogurt with a tsp sugar and 2 slices of bread.
Have you ever heard of Jessie Inaupsché? => Dress your carbs! And eat by listening to your body sensations. If you're still / if your liver is still recoverable. -
RE: Canned fish and seafood
hi,
When eating canned fish or pre-packed meat, we need to take the amount of histamine into account. Not only inside (H3 for tuna) but the effect on mastocytes (L3). L stands for liberation (what makes it free). Tuna content is 600 Unities. We can manage 60 - 100 U per day.
Mackerel contains far less.
For PUFA, as long you stay under 1.5 gr omega-3 the body can manage. I target max 6 gr LA, on a usual take.
You can combine and take antihistamines to fight symptoms for a certain period. There, you calm the game in the mastocytes (stabilizer => brake L1-3). Histamine is released from mastocytes or basophiles under the influence of allergens. The DAO (Diamine Oxidase) route can require additional B6, magnesium and copper intake. But beware, it is not appropriate to take 1.5 mcg of Cu bisglycinate every day (overload). To assist, an anhydrous quercetin treatment is strongly advised, in attack, to calm the mastocytes (cells that release histamine). Bromelaine is often combined with quercetin and dioic nettle in anti-allergic products. But you only calm a way ...
Useful link (in french, translator needed) if you deal with allergy and want to take a supplement (DAO)
DAO pour intolérance à l’histamine
https://mirzoune-ciboulette.forumactif.org/t1745-traiter-lintolerance-a-lhistamine#28835 -
RE: Healing ups and downs - PEM query
@jd_au said in Healing ups and downs - PEM query:
Update: I now don't think today's 'crash' was PEM, but rather a paradoxical reaction to the thiamine. Even at low doses (I've been taking about 50 mg hcl),
Hi,
With thiamine high dose you need some B2 to avoid side effects. A significant dose of thiamine will exhaust vitamin B2 (Riboflavin). It is as if there were then a lack in Riboflavin.
When taking B1 high dose there is communication between the brain and the body through the vagal nerve (and thus the microbiome).
Mostlylurking has seen side effects when taking high dose Thiamin (B1) without supplementing riboflavin (B2). Not necessarily every day. Marked temporary increase of riboflavin excretion was not observed by all subjects (period not above 8 days).
Riboflavin deficiency (B2) can have some of its effects by reducing the metabolism of other B vitamins, including folate and vitamin B-6.PS: I confirm there are up and down levels when the brain is trying to adapt the response for energy.
useful info (in French, translator needed, with link in English)
*) Thyroïde B1 B2 B3 – Interaction
Interaction entre les vitamines du groupe B et la thyroïde
https://mirzoune-ciboulette.forumactif.org/t2018-comment-booster-votre-energie#29549
Excerpt:
Metabolism of B2
Vitamin B2 (Riboflavine), in its forms of coenzyme, Riboflavine-5′-Phosphate (FMN) and Dinucleotide Adénine Flavine (FAD), is involved in many critical metabolic reactions, including the assimilation of amino acids, carbohydrates and lipid metabolism, and in the conversion of folic acid (B9) and pyridoxine (B6) in their forms in coenzyme.
The enzymes for the conversion of the FAD and the FMN will be regulated by the Thyroid T3 and T4.
Expressed differently:Thyroid hormone (thyroxine) regulates the enzymatic conversion of riboflavin into its active coenzyme forms.
B2 is necessary for the regulation of thyroid enzymes: B2 helps to organify iodine (i.e. the incorporation of iodine for the production of thyroid hormones). (5-9)
-
RE: What Did Dr. Pete Say The K-2 To Calcium Ratio Was?
@Amethyst said in What Did Dr. Pete Say The K-2 To Calcium Ratio Was?:
So how much to take if I am taking in at least 1,200 mgs. of calcium?
How much vitamin K?
I’ve had a discussion on RPF about the dosage of vitamin K2. How much is too much?
Details are given beneath (last part).
I won’t recommend taking more than 2 mg / day, given the fact that the menaquinones (K2 family) are broken down into menadiones, which can be "toxic" If repeated.
http://ajplung.physiology.org/content/ajplung/262/5/L637.full.pdfHere are the main points
Haidut said:
“Does it mean that more than 2mg K2 at a time will lead to toxic levels of K3?”
LucH answered:
Well, we don't know exactly. This is a supposed by-side effect. I explain.
Conversion of excess menaquinones in menadiones
We know menadione is a metabolite of oral vitamin K supplement.
Phylloquinone (K1) is converted into menaquinone-4 and accumulates in extrahepatic tissues. Neither the route nor the function of the conversion is totally known. One supposed metabolic route is the conversion of 5 – 25 % of menadione from phylloquinone by catabolic activity. 10 – 15 % is most likely converted. But this is aleatory (due to intestinal absorption) and not equally distributed. We can only measure the presence of menadione in urine and make deductions when excreted. (1)
When taking excess menadiones, through conversion / excretion it would be quite advisable to take some NAC to get enough selenium to neutralize ROS. Adaptive response. (2) Menadione generates ROS through redox cycling, and high concentrations trigger cell death. If lack of glutathione. (3 – 4)
Edit: No NAC if supplementing B3 vitamin.
Correction form Haidut:
Haidut said:
I think selenium is a much safer option then NAC. The cysteine has a lot of bad effects on the organism.
LucH answers:
=> Yes, cysteine could depress thyroid function in case of insufficient iodine.
Conversation following, by LuH:Menadione in excess clearly affects the mitochondrial function. (5)
However there is no reported health risk when taking K2. Excess K2 is excreted through urine and feces (through bile). No direct adverse effect doesn’t mean there is no impact on energy level. This is a personal comment. It depends on the way the body neutralizes an excess oral supplement.
Mind excess alpha-tocopherols too (vitamin E) because it may interfere with synthesis of K2. (6- 7) Also if you take phytonutrients as AINS (curcumine or aspirin) because it affects cytochrome P450 (detox pathway). You need a recovery period between 2 cures.- https://www.researchgate.net/publication/7305589_Menadione_is_a_metabolite_of_oral_vitamin_K
- http://www.jbc.org/content/281/52/40485.full.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3005834/
Oxidative Stress-Induced Apoptosis by Menadione at high concentration. - https://www.ncbi.nlm.nih.gov/pubmed/20937380 +
- http://www.sciencedirect.com/science/article/pii/S1521661601951290
- http://jcs.biologists.org/content/106/1/309
- http://health.oregonstate.edu/biblio/vitamin-e-decreases-extra-hepatic-menaquinone-4-concentrations-rats-fed-menadione-or
- http://www.pubpdf.com/pub/22707266/Vitamin-E-decreases-extra-hepatic-menaquinone-4-concentrations-in-rats-fed-menadione-or-phylloquinon
α-Toco may interfere with the side chain removal of phylloquinone (PK) to form menadione (MN) as an intermediate for synthesis of tissue-specific menaquinone-4 (MK-4).
Paymanz said:
“you didnt meant to say alpha tocopherol inhibit its conversion ,right?”
LucH answers:
=> Tocopherols neutralize oxidant molecules. If excess menaquinone leaves quinones, if menadione generates ROS, yes, alpha tocopherols would serve as firemen. Thus tocopherols could inhibit K2 when taken at the same time. Only a supposition but it’s logical.More info to optimize Ca and phosphorus (between 2.2/1)
Bone metabolism and joints: Dependent on vitamin K2
To absorb Ca (via osteoblasts), the body is dependent on vitamin K2. The synthesis of osteocalcin depends on vitamin K2. If we schematize: osteocalcin allows the body to use calcium for bone metabolism. In the absence of vitamin K, osteocalcin cannot function. Vitamin K2 is necessary to carboxylate/activate K2-dependent proteins so that they perform their functions. For example, the matrix Gla protein (MGP protein or osteocalcin), it prevents calcium from being deposited on blood vessels and other soft tissues. The more vitamin K2 you lack, the less MGP will be activated, so calcium will lodge in the arteries, creating stasis and then blockage... NB: the matrix Gla protein is not specific to bone: it is also found in the wall of vessels where it plays a role in inhibiting vascular calcification.See animation on this link (bone remodelling):
http://www.random42.com/bone-biology-osteoblasts-and-osteoclasts-animation
Info on absorption, target and dosage of vitamin K (and links from Chris Masterjohn and Kate Rheaume-blue) (in French, with English links)
https://mirzoune-ciboulette.forumactif.org/t1704-vit-k-mk4-vs-mk7-update-02-2020#21497Momado965 said:
Do you think a dose of 45mg of mk4 spread throughout is toxic?
Luch answered:
Toxic, no.
K1= 45 mcg.
Enough to improve the level of artery calcification and cardiac mortality. It depends on the interaction between A D K vitamins.
K2 type MK4: 45 mg (45.000 mcg).
Recommended to decarboxylate the Osteocalcin level.
K2 type MK7: 240 mcg for diabetics and obese people. Level recommended in the prevention of atherosclerosis, diabetes, osteoporosis, obesity, etc.
From « Dosage optimum » on this link (in French):
http://mirzoune-ciboulette.forumactif.org/t706-vitamine-k2-achat-exemples-de-produits#6433 -
RE: Beef gelatin
@Cezar4911 said in Beef gelatin:
Would buying cheap beef gelatin powder in bulk be problematic because of endotoxins?
Try to buy one with certificate, not the cheapest but at an interesting price. If you buy the lowest, without guarantee, you throw your money out. Or Make your own collagen bone broth.
You can buy powder from Amazon. A well-known vendor of gelatin is purebulk.com. -
RE: Methionine/Cysteine restriction increases longetivity AND energy expenditure
@Mauritio said in Methionine/Cysteine restriction increases longetivity AND energy expenditure:
anybody tried sodium selenite and selenomethionine and can compare them ?
*) Forms of sélénium
The forms of selenium exist in 3 major forms:- Yeast
- Sodium (selenite or selenate)
- Amine acid (Selenio-Methionine or Cysteine).
Selenate or seleno-methionine (SM) are generally found on the market. Specialists classify the Se in two main categories: organic and inorganic. The inorganic salts would be supposed to be less well absorbed by the tissues. If we only refer to SM and Selenate, this is not really true, as several studies have shown it. (4)
*) Biodisponibility
The degree of absorption of selenite is less, but sufficient, of the order of +/ 50 to 60 % against 75 % for selenomethionine. (2)
What form is the best?
Selenium in any form whatsoever, that it comes from food or supplements, organic or inorganic, is used by the body for the synthesis of selenoproteins after being first metabolized in hydrogen selene, selenium cellular storage. The surplus of selenium is converted into methylated metabolites and excreted by urine and breath. Excessive accumulation of hydrogen selenial can cause its oxidation, resulting in the production of reactive oxygen species (ROS) resulting in oxidative toxic effects in the body. (o)
All forms of selenium are well absorbed, but the absorption of selenomethionine is the best. They use the same active transport mechanism as for methionine, one of the 9 essential amino acids which can only be obtained by food, which increases the effectiveness of absorption of selenomethionine compared to the inorganic forms of the selenium.
*) Recommended Daily Supply
Currently recommended nutritional supply (RDA) in adults is 55 micrograms/day. - Daily needs are 55 mcg (AJR). 1 mcg /kg of weight is optimal.
- Diet (1) provides around 50 % of needs if you eat meat or legumes. A single nut from Brazil / Amazon covers needs (…).
- In the event of pathology / detox, 200 - 300 mcg can be prescribed punctually.
- “Too Much of A Good Thing is Bad!”
=> Selenium supplementation should not be taken every day (half-life). Except in particular cases, in the event of a detox for example, therefore punctually (2 to 300 mcg per day, in detox cure). But in this case, a contribution of curcumin would be desirable to reduce hepatic toxicity due to the impact on dehydrogenase enzymes. - Taking different forms of selenium is interesting, especially in the event of cancer prevention (recurrence).
- If you exceed +/ 100 mcg/ L in the blood serum, you impact the operation of certain dehydrogenase enzymes, especially required in the Krebs cycle.
*) More details on this post, with sources and references.
Selenium: Too much of a good thing is bad. Forms and enzymes
https://mirzoune-ciboulette.forumactif.org/t1953-selenium-too-much-of-a-good-thing-is-bad#27971 (in French ) translator needed but with references from studies in English)
Sources et Références
0) Toxicité hépatique due à l’impact sur les enzymes déshydrogénases
Rôle protecteur de la curcumine
Protective effect of curcumin during selenium induced toxicity on dehydrogenases in hepatic tissue. 2005
PMID: 15881869
Selenium administration resulted in a marked decrease in the activity levels of the liver succinate dehydrogenase, malate dehydrogenase, and lactate dehydrogenase while pyruvate dehydrogenase increased significantly.
2) http://www.hc-sc.gc.ca/ewh-semt/pubs/water-eau/selenium/index-fra.php
Avec références d'études. -
RE: What Did Dr. Pete Say The K-2 To Calcium Ratio Was?
@Amethyst said in What Did Dr. Pete Say The K-2 To Calcium Ratio Was?:
Anyway, back to my question. If you are consuming a lot of dairy ( which I am now) how much K-2 does one supplement with?
Hi,
You have to decide which king of K2 you want: MK4 or MK7?
Mk4 twice a day (half life); MK7 must be protected from oxidation (no liquid form). -
RE: When peating fails
@himblondemaxxing said in When peating fails:
Feel like I am getting worse and more hypothyroid which is discouraging... can anyone else relate?
Hi,
I’d have a look at these points to deal with anti-energy problem (part of the whole point)- Estrogen imbalance
- Thyroid saturation by heavy metals
- See Kate Deering too to optimize the metabolism.
Note: It’s not because RP said sth was good in general that you don’t have to manage according to your ground and the level of you energy (the way you metabolize).
Useful info:
*) The link between thyroid, energy and excess estrogen.
Statistics show that one in eight women between the ages of 35 and 65 and one in five women over the age of 65 have some form of thyroid disease. If T3 is impaired, the neurotransmitter will fail to transmit the right impulse to regulate the action of serotonin, norepinephrine, and GABA (gamma aminobutyric acid). If you don’t have enough T3, or if its action is blocked, an entire cascade of neurotransmitter abnormalities may ensue and can lead to mood and energy changes, including depression.
Source: Effects of estrogen on thyroid function
http://www.drnorthrup.com/thyroid-disease/*) Iodine deficiency is also very common in women.
See Thyroid Toxicity, By Philip L. Roberts, MD- What intoxicates the thyroid?
-- Watch out fluorine and bromine: they saturate the receptors of the thyroid and prevent iodine from fixing / to reach the target. - Useful nutrients for the thyroid.
Les vitamines liposolubles : A, D, E, K.
Les vitamines C B-2 B9 et B12
Les oligo-éléments : Magnésium – Sélénium – Zinc – Iode
More info in the book « Stop the thyroid madness II, chapter 10.
How Thyroid Experts are Challenging Ineffective Treatments and Improving the Lives of Patients
JANIE A. BOWTHORPE, M.ED. EDITOR.
Or a summary of the book on my forum:
http://mirzoune-ciboulette.forumactif.org/t119-the-thyroid-madness#817
*) Brain protection from aluminum, cadmium, lead, mercury and nickel.
Magnesium protects the brain from the onslaught of toxic minerals like aluminum, cadmium, lead, mercury and nickel. Low levels of brain magnesium allow the toxic metals to enter brain cells, and could be the harbinger of conditions like Alzheimer’s and Parkinson’s
We recommend combining ionized magnesium with choline citrate to enhance magnesium uptake and chaperone its delivery to cells.
Regular magnesium is often not absorbed efficiently, especially in people with compromised digestion and chronic conditions; these are the people who most need the support that magnesium provides. The cellular uptake of magnesium entails an energy-dependent step that is bypassed by the addition of choline.
Source: Three Safe & Simple Ways to Detox
Note: If you suffer from mercury toxicity, any recovery can’t take place. There is a strict protocol to follow. And mind current mistakes: too long with the same product. No recovery stage allowed for energy. Get informed on the health crisis before detoxifying.Useful info:
- Book: How to heal your metabolism.
http://mirzoune-ciboulette.forumactif.org/t1347-comment-guerir-votre-metabolisme-livre
=> Large excerpts on my forum (in French). Top 3 of the read posts. - Mind ratio Hg/ Se in fish and seafood. Selenium (Se) must be higher than mercury (Hg)
Ratio Hg / Se mg / 100 gr fish
Espadon 0,89 / 0,062 mg / 100 gr de poisson (swordfish)
Maquereau espagnol 0,44 / 0,36 (Spanish mackerel)
Saumon d'élevage atlantique 0,06 / 0,041
Saumon sockeye d'Alaska 0.03 / 0,030
-
RE: Histamine Intolerance?
@Lothric said in Histamine Intolerance?:
Does anyone have any idea the cause or what to look for here? Is it histamine? Estrogen? Serotonin? The proximity to her menstrual cycle makes me think it’s hormonal.
Hi,
I've been dealing with histamine allergy + suspicion of SIBO/SIFO. Under control but still managing the problem.
I can give a link to my log on my forum if wanted.
The problem is complex:- not enough enzymes to get rid of excess histamine.
- Time-delay to evacuate histamine.
- know your enemy: Not only H3 (bringing high histamine) but also L3 (free histamine liberated in reaction to some stuff). When the liver has to deal with excess amines, there is overload.
I've got a pdf doc with info (H3 and L3). - Appropriate microbiote after having taken antibiotics or if suffering from dysbiosis.
Note: cypro will act as anti-histamine of first generation but it won't solve anything, only telling the mastocytes to react less. Useful at the beginning, but not solving anything.
-
RE: moderate protein restriction can cure all ills
@LetTheRedeemed said in moderate protein restriction can cure all ills:
What? Are you saying if you eat more carbs with meat, you’ll lack immediate fuel?
No, WE need some carbs when eating meat. Not much required. A tomato or an orange IS fine. I Can show it with a graph if desired.