@A-Former-User said in Restoration of the Biofield topic:

@ThinPicking I'll try to explain:

I posted the original post to this thread. I mentioned "biofield" and I also wrote about Dr. Robert Young. @yerrag responded that yes, the biofield is a real thing. @yerrag also said that Dr. Robert Young is not a credible source and listed reasons why. I responded to @yerrag and wrote a little about the biofield/aura/chi. I also wrote that I agree with @yerrag that Dr. Robert Young is not a good source and that I regret that I linked to his site. Note that I posted several times at the beginning of this thread and have provided multiple links to sources other than Dr. Robert Young in these posts. I could have and should have simply left Dr. Young out of the topic entirely for the sake of clarity.

Dr. Robert Young makes some good points on his blog and in his videos. However your own discernment abilities must be activated when reading/watching him. Simply opening up the top of one's head and pouring Dr. Young's (or anyone else's) ideas in unabated can be hazardous to one's own well being. Discernment abilities can be thought of as "intuition" or "spidey senses" which ties back into qualities of an activated biofield and why it is important to have one.

Ray has mentioned something similar to a biofield, although it is more about how health practitioners in the middle ages (I would associate the notable ones like Avicenna or Ibn Senna) approach healing in a way that isn't mechanistic. This aligns a lot with the thinking of Tom Cowan even, as he is influenced as much in his thinking by Rudolf Steiner. These names are outside the western approach to healing. Even in his book Mind and Tissue, which I only read halfway through, Ray talks as much about the 'active field' which is more compatible with the Eastern mind (Eastern as in Greek and Russian orthodoxy) which is less influenced by the materialistic philosophy of Descartes, which I gather puts forever into the Western educated mind a reductionistic approach to seeing things.
And this is clear when you waddle in between hospitals and alternative healers, and find experts talking over each other because of the philosophical divide.

@haidut I suspect that 5-HT is high in babies because constipation/impaction of the gut would be life threatening for a baby. 5-HT keeps things moving along in the intestine; it increases/intensifies peristalsis.
Serotonin Deficiency Is Associated With Delayed Gastric Emptying

I suspect that the issue of serotonin (5-HT) getting through the intestinal wall and into the blood stream is the primary concern; so long as the 5-HT remains inside the intestine (along with the endotoxin) things should be fine. If the integrity of the intestinal wall is compromised and endotoxin and 5-HT are able to seep through into the blood stream major problems happen (sepsis, high serotonin symptoms).

The health of the epithelial cells that line the intestine are important because if they fail, leaky gut happens. Thiamine is believed to be important for the epithelial cell function. See here: Dietary supplementation of thiamine enhances colonic integrity and modulates mucosal inflammation injury in goats challenged by lipopolysaccharide and low pH "The results show that dietary thiamine supplementation could improve the colon epithelial barrier function and alleviate mucosal inflammation injury in goats after lipopolysaccharide and low pH challenge."

Another important body part that requires excellent epithelial cell function is the blood/brain barrier. Thiamine deficiency compromises the blood/brain barrier too.

Thanks for all you do! I always appreciate your posts.

@haidut is leucomethylene blue safe to take? It is what is left over after turning ascorbic acid ----> dehydroascorbic acid with MB. You've said before the mixture can be left out after the LMB and DHAA are produced and the LMB will oxidize back to MB, yet I'm unsure if the DHAA degrades in the meantime. I've thought of bubbling air back into the solution to get it to go blue again. And then again, I have the suspicion that the redox state of MB/LMB is pretty much inconsequential as it cycles between the forms relatively rapidly once ingested. My urine still changes color wether I take MB by itself or LMB + DHAA.

I like taking it during winter.

No thanks Lurking.

@SpicyPeater said in screenshot of T3 Uncoupled post RE Coffee 📉testosterone:

https://u.pcloud.link/publink/show?code=XZMyuc0ZdjYs4iDfpEBp7h6mvqwkxRQzJHKX

Reductionist BS...aka Twitter.

@pannacottas No but Ive tried memantine, and I prefer to amantadine on every metric except the long half life (makes sleep impossble for me). Bromantane might be my saving grace though since its half life is not as much and I heard is more dopaminergic than either memantine or amantadine.

@calvinklein21101 said in Vitamin A may prevent/treat motor neuron diseases, including the lethal ALS:

something going wrong in their metabolism

I read Poisoning for Profits and my first inclination wasn't to start rattling on about my liver while chomping rice and beans all day. Something about it seems to ring with them.

@Serotoninskeptic

The values above need to be revised. The doses of calcium needed in practice to complex with oxalate are higher than expected, and how calcium interacts with phosphate is relevant:

"The degree of interference reported here (~166 mg phosphorus for every 500 mg ingested calcium—0.4 mmol phosphorus for every mmol calcium) is substantially less than might have been predicted from simple stoichiometry. A 1:1 molar ratio for CaHPO4 would predict binding of 388 mg phosphorus by 500 mg calcium and, for Ca3(PO4)2 at a Ca:P molar ratio of 1.5:1.0, 258 mg phosphorus would be bound per 500 mg calcium. A molar ratio of 1:1 seems more likely at digestate pH values below 7.0. Even at the upper end of the confidence interval for the regression model—210 mg (6.8 mmol)—binding would still be substantially less than the lowest stoichiometric prediction. This discrepancy may be partly due to rapid absorption of phosphorus in the duodenum well before calcium and phosphorus complexes can form in the chyme. In any event it undoubtedly reflects the complexity and multiplicity of the interactions within the digestive residue."

(10.1080/07315724.2002.10719216)

Compare them:

Predicted: Ca 1:1 P Practical: Ca 2.5:1 P

For oxalate:

Predicted: Ca 1:1 Oxa Practical: Ca ?:1 Oxa

If we treat the interaction with oxalate as that with phosphorus (2.5:1) and adjust for mass (2.5:2), we would need 125 mg of calcium for every 100 mg of soluble oxalate.

One problem is guessing how much is soluble from the total oxalate content of a food. Based on the last two 'Cooked' columns (⇈), it's all over the place.

Another problem is the additional confounders. Member 'blabla' posted this elsewhere:

cab6428c-10ca-4047-a0e4-08d21350c5b1-image.png
⠀(10.1097/01.asn.0000127864.26968.7f)

Up to 800 mg: dietary calcium alone More than 800 mg: dietary calcium and supplements (400 mg and 1000 mg)

Only the supplements were dosed with the labeled oxalate, suggesting that indirect interference is possible, but notice the degree of variations at low intakes. If you must supplement, it's preferable to dose in the same meal:

edacf8d3-917e-41a6-b44f-6a24525a3abb-image.png
⠀(10.4065/79.1.91)

But when the diet contains enough calcium (1000 mg), adding more seems unnecessary because a further reduction is minimal.

@thyroidchor27

P5P can help to reduce homocysteine.

@mikeyd

So if you can’t tolerate wheat and have SIBO, what foods would you recommend? I too, have issues with many fruits. I try to peel the skin off of most fruits before I eat them. I think this helps but I still feel inflamed. I skin dates, peaches etc. I can’t eat apples or pears at all. Please help, like you I’ve had it probably for 10 years but only heard of term SIBO this year.

Elevated fructose in the brain is a sequester to protect it because of AD. Much like the protein plaques, which have been erroneously blamed as a result of AD.

The rationale was: carbon dioxide goes through cells' lipids and releases a proton inside once hydrated. Then, let's find another compound that's also not barred by lipids and releases even more protons. This led to 2,6-DHBA.

It overlooks that the very property that makes the selected compound a greater acidifier is also what makes it extraordinarily prone to deprotonate prematurely. The ability to pass through lipids is likely to be compromised with the early proton release. If it could be injected into the tumor, it would still be uncertain if it could be taken up by target cells.

Healthy Cancerous Extracellular pH 7.4 ↓6.5 Intracellular pH 7.1 ↑7.5

For direct internal acidification, it would be more fitting to seek weak rather than strong acids, those that can take advantage of the bump in transitioning from the extracellular to intracellular compartment (6.5↗7.5) to deprotonate the molecule where intended.

Relevant for many and thank you as always.

This info will be passed on.

Also, if b.i. progesterone actually maintained or reduced the size of a meningioma.

Hello, Jorge (or your bot).

That these vitamins increase the risk of cancer complications is not a reason to avoid supplementation, but to be more cautious with them.

One suggestion was to offer B-vitamins in separate products, something like:

Mitochondrial function and toxicity: role of the B vitamin family on mitochondrial energy metabolism Mitochondrial function and toxicity: role of B vitamins on the one-carbon transfer pathways

But it's challenging to have a clear separation of vitamins because of interrelationships. Pyridoxin was excluded from their 'energy metabolism' group, but it's directly involved in glycogen use and the transaminases to prepare amino acids for oxidation. Biotin was included, but associated with anabolic functions, and one of the catabolic pathways would be dependent on the vilified (adenosyl)cobalamin. If all came down to an energy problem, the nucleotides of the energy-releasing coenzymes involve folate in their syntheses.

Cancer Cells Tune the Signaling Pathways to Empower de Novo Synthesis of Nucleotides

0fa521f5-78b7-4401-88c7-079d8641aa8d-image.png

Growth-promoting substances don't just fuel cancer, they're also needed for cell and tissue regeneration.

Supplementation is discouraged for folate, cobalamin, and choline (or betaine). We know that the message is reaching a community that doesn't consume a lot of leaves (foliage→folate), nor legumes. The availability of folate in oranges is arguably lower than expected.

Properties of Food Folates Determined by Stability and Susceptibility to Intestinal Pteroylpolyglutamate Hydrolase Action

Tipping the reliance in favor of folate-cobalamin over choline might yield extra glycine, that can be formed for every folate cycled. The simultaneous restriction of these vitamins, along with a predisposition to their insufficiency, malabsorption syndromes, and the adoption of a diet low in methionine doesn't seem promising.

Do we have indications that the population is adequate in the anabolic vitamins and can afford a restriction? Because the typical consumption and positive reactions to supplementation suggest otherwise.

Not only a deficiency of any vitamin tends to predispose to cancer, but cancer is not the only disease to worry about. Cardiovascular problems and infections are major threats.

Digestion weakens in advanced age and disease, making it helpful to have available these vitamins in purified form. The issue here is that you're leaving for other vendors to do the dirty job of guaranteeing adequacy in a considerable number of people that would benefit from the carcinogenic nutrients.

Thank you for sharing this interesting new angle. It fits very well with the recent interest in the connection between antibiotics and AD. In the last three years, several large studies looking at the efficacy of antibiotics have been started, and they should probably yield some very interesting results. In the end, it all comes down to your gut and endotoxins.

@haidut said in Pregnenolone (P5) may treat for COVID-19, by increasing dopamine:

Given that the mechanism of action reported by the study (dopamine enhancement) is so generic, the findings of the study possibly apply to other respiratory viral infections (URTI) too

Dopamine was quite important in renal function the last time I checked, renal function quite important to the RRAS, the RRAS quite important to the "spike protein" (an ACE2 inhibitor, allegedly).

It's highly likely to me that it does apply to other URTI. In which case I wonder why we're using the word "covid". Other means to walk believing masses back are available.

@stag

I wonder if their theories can be shown on blood tests.
Although I know how Dr. Peat feels about free T4 and free T3 labs, an rT3 might be good to track in such cases.

My rT3 indeed does go down when I increase my T3 while taking T4/T3.

Thank you for sharing this - I will look into their therapies further.