@Amazoniac said in Toward a Better Understanding of Reactive Oxygen Species:
The ROS linked to fat metabolism can arise partly from parallel effects, such as structural or functional perturbations and up-regulation of alternative ROS-producing enzymes (NOX, XO, COX, LOX, CYP2, etc.).
I suppose that here we're mainly talking about excess PUFA loaded in adipocytes (AA cascade). When we stress or have a diet (fat loss).
If we remain under 10 g PUFA/day, preferably 5-6 g, it would be OK.
Edit: fine, the picture on ROS effects. (downloaded)
ROS concentration & deleterious effects on cells.
The next question is: What about when corn / soy food (from real food) is eaten, with LPS by-side load. Otherwise, it remains a theory. 1° In presence of ALA (thiol antioxidant) or a lipoprotection (A D3 K2 + vit E).
2° The same with aspirin or WWB.